Cardiovascular · PANCE / PANRE

Rheumatic Fever and Rheumatic Heart Disease

Post-streptococcal autoimmune sequela causing pancarditis acutely and chronic valvular damage (mitral > aortic) years later.

Also known as: ARF, acute rheumatic fever, RHD, rheumatic heart disease, Jones criteria

Overview

Acute rheumatic fever (ARF) is a delayed, immune-mediated inflammatory disease following untreated group A streptococcal (GAS) pharyngitis, characterized by carditis, arthritis, chorea, and skin findings. Rheumatic heart disease (RHD) refers to the chronic valvular sequelae, classically progressive mitral stenosis, that develop years to decades after one or more episodes of ARF.

Epidemiology

Rare in industrialized nations but a leading cause of cardiovascular morbidity in low-resource regions; >40 million people live with RHD worldwide. Peak incidence of ARF is 5-15 years. Mitral valve is involved in roughly 70% of RHD cases; combined mitral and aortic involvement in ~25%.

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Risk factors

  • Untreated or undertreated GAS pharyngitis (Streptococcus pyogenes)
  • Crowded living conditions, poverty, limited access to care
  • Indigenous populations (e.g., Pacific Islander, Aboriginal Australian, sub-Saharan African)
  • Family history of ARF/RHD (HLA association)
  • Age 5-15 years for primary attack; recurrences may extend into adulthood

Pathophysiology

Molecular mimicry between GAS M-protein and human cardiac myosin, valve glycoproteins, and neuronal antigens triggers cross-reactive antibody and T-cell responses. Acutely, this produces pancarditis (endocarditis, myocarditis, pericarditis), arthritis, and CNS inflammation. Chronic, repeated bouts of valvulitis cause leaflet thickening, commissural fusion, and chordal shortening, producing the characteristic fish-mouth deformity of rheumatic mitral stenosis.

Clinical presentation

Symptoms

  • Migratory polyarthritis of large joints (knees, ankles, elbows, wrists) — exquisitely responsive to salicylates
  • Carditis: new murmur, chest discomfort, dyspnea, fatigue, palpitations
  • Sydenham chorea: involuntary purposeless movements, emotional lability, milkmaid's grip — appears weeks to months after pharyngitis, may be the sole manifestation
  • Fever, malaise, anorexia following pharyngitis 2-4 weeks earlier
  • In chronic RHD: progressive exertional dyspnea, orthopnea, palpitations from AFib, hemoptysis

Signs / physical exam

  • New apical pansystolic murmur (mitral regurgitation), Carey Coombs mid-diastolic murmur, or early diastolic murmur of AR
  • Erythema marginatum: evanescent, serpiginous, non-pruritic pink rings on trunk
  • Subcutaneous nodules: firm, painless, over extensor surfaces (rare, severe disease)
  • Pericardial friction rub or effusion in carditis
  • Chronic RHD: opening snap and rumbling mid-diastolic murmur of MS, irregularly irregular pulse (AFib)

Classic findings

Migratory polyarthritis + new murmur + recent pharyngitis = ARF until proven otherwise. Erythema marginatum and subcutaneous nodules are highly specific but uncommon.

Differential diagnosis

  • Reactive arthritis (post-streptococcal arthritis without other Jones criteria) — Persistent migratory or additive arthritis with minimal response to NSAIDs; no carditis or chorea; ASO titer elevated but lacks Jones threshold
  • Juvenile idiopathic arthritis (JIA) — Insidious onset, morning stiffness, may have uveitis; arthritis pattern is symmetric and chronic, not migratory; negative ASO
  • Lyme carditis / Lyme arthritis — Tick exposure, erythema migrans, AV block; Borrelia serologies positive
  • Infective endocarditis — Persistent fever with positive blood cultures and embolic phenomena; vegetations on TEE
  • Systemic lupus erythematosus (Libman-Sacks endocarditis) — Multisystem disease with ANA, anti-dsDNA, malar rash; nonbacterial verrucous valvular lesions
  • Sydenham chorea mimics (Huntington, Wilson, drug-induced) — Family history, KF rings, ceruloplasmin levels; medication exposure (neuroleptics, stimulants)
  • Viral myocarditis — Recent viral prodrome, no migratory arthritis or chorea; elevated troponin and global LV dysfunction

Diagnostic workup

Diagnostic criteria

2015 Revised Jones Criteria (AHA): initial ARF requires 2 major OR 1 major + 2 minor, with evidence of preceding GAS infection. Recurrent ARF allows 3 minor in addition. MAJOR (low-risk populations): carditis (clinical or subclinical), polyarthritis, chorea, erythema marginatum, subcutaneous nodules. MINOR: polyarthralgia, fever ≥38.5°C, ESR ≥60 or CRP ≥3, prolonged PR. Moderate/high-risk populations have less stringent major criteria (monoarthritis or polyarthralgia can count, fever threshold ≥38°C).

Labs

  • Throat culture or rapid antigen detection (often negative by time of ARF presentation)
  • Anti-streptolysin O (ASO) titer — rises 1 week, peaks 3-6 weeks; combine with anti-DNase B for sensitivity >95%
  • ESR and CRP (elevated; used as minor criteria)
  • CBC (mild leukocytosis), BMP
  • Blood cultures to exclude infective endocarditis

Imaging

  • Transthoracic echocardiography in all suspected cases — detects subclinical carditis (mitral regurgitation jet, leaflet thickening); now incorporated into 2015 revised Jones criteria
  • 12-lead ECG: prolonged PR interval (minor criterion), AV block, AFib in chronic RHD
  • CXR: cardiomegaly, pulmonary congestion if HF present

Diagnostic algorithm

ManifestationCategoryNotes
Carditis (clinical or subclinical)MajorMost consequential — drives long-term morbidity
Polyarthritis (or monoarthritis in high-risk)MajorMigratory, large joints, exquisitely aspirin-responsive
Sydenham choreaMajorLatent period months; may be sole feature
Erythema marginatumMajorEvanescent, non-pruritic, trunk and proximal limbs
Subcutaneous nodulesMajorPainless, extensor surfaces, severe disease
Polyarthralgia (low-risk) / Fever ≥38.5°CMinorCounts only if not used as major
ESR ≥60 or CRP ≥3.0 mg/dLMinorAcute-phase reactants
Prolonged PR intervalMinorNot counted if carditis is major
2015 Revised Jones Criteria for diagnosis of initial acute rheumatic fever (low-risk populations).

Treatment

First-line

  • Eradicate streptococcal infection: penicillin G benzathine 1.2 million units IM once (or 600,000 units if <27 kg); alternative oral penicillin V or amoxicillin × 10 days; azithromycin or cephalexin if penicillin allergic
  • Anti-inflammatory for arthritis and mild carditis: high-dose aspirin 50-100 mg/kg/day divided QID (taper over weeks based on symptoms/inflammatory markers); naproxen is increasingly used as an alternative
  • Severe carditis with heart failure: corticosteroids (prednisone 1-2 mg/kg/day, taper over 2-4 weeks) plus standard HF therapy
  • Bed rest until inflammation subsides; gradual return to activity

Secondary prophylaxis (essential after ARF)

  • Penicillin G benzathine 1.2 million units IM every 4 weeks (every 3 weeks in high-risk regions)
  • Alternatives: oral penicillin V 250 mg BID, sulfadiazine, or macrolide if allergic
  • Duration: ARF without carditis — 5 years or until age 21 (whichever longer); ARF with carditis, no residual disease — 10 years or until age 21; carditis with residual valvular disease — 10 years or until age 40 (lifelong if recurrent or severe)

Second-line / adjunct

  • Sydenham chorea: usually self-limited; for disabling symptoms use carbamazepine or valproic acid; haloperidol historically used but more side effects
  • Heart failure management: loop diuretics (furosemide), ACE inhibitor (enalapril, lisinopril), beta-blocker once stable
  • Valve surgery (balloon valvotomy, repair, or replacement) for severe symptomatic RHD

Complications

  • Chronic valvular disease, especially rheumatic mitral stenosis (most common chronic sequel)
  • Atrial fibrillation with thromboembolic events (stroke, systemic emboli)
  • Heart failure, pulmonary hypertension
  • Infective endocarditis superimposed on damaged valves
  • Recurrent ARF without adequate secondary prophylaxis

PANCE pearls

  • Jones criteria require evidence of preceding GAS infection EXCEPT in chorea or indolent carditis, where the latent interval can be months.
  • Throat culture is often negative by the time ARF develops; rely on ASO and anti-DNase B titers.
  • Aspirin classically rapidly resolves arthritis within 48 hours; failure to respond should prompt reconsideration of the diagnosis.
  • Subclinical carditis (echo-detected MR without auscultatory findings) now counts as a major criterion in the 2015 update.
  • Rheumatic mitral stenosis is the prototypical cause of MS worldwide and characteristically has an opening snap with mid-diastolic rumble and presystolic accentuation if in sinus rhythm.

References

  • AHA 2015 — Revision of the Jones Criteria for the Diagnosis of Acute Rheumatic Fever in the Era of Doppler Echocardiography (Gewitz et al., Circulation 2015)
  • AHA/ACC 2020 — 2020 ACC/AHA Guideline for the Management of Patients with Valvular Heart Disease (Otto et al., Circulation 2021)
  • WHF 2012 — World Heart Federation Criteria for Echocardiographic Diagnosis of RHD (Reményi et al., Nat Rev Cardiol 2012)
  • IDSA 2012 — IDSA Clinical Practice Guideline for the Diagnosis and Management of Group A Streptococcal Pharyngitis (Shulman et al., CID 2012)

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