Anterior Cruciate Ligament (ACL) Tear
Non-contact pivoting injury with audible pop, immediate large effusion, and knee instability; reconstruction in active patients.
Also known as: ACL tear, anterior cruciate ligament tear, ACL rupture, ACL injury
Overview
Partial or complete rupture of the anterior cruciate ligament, the primary restraint to anterior tibial translation and a secondary restraint to rotational laxity. Usually a non-contact injury during deceleration, cutting, or landing.
Epidemiology
Estimated 200,000 ACL injuries annually in the US. Most common in athletes aged 15-25. Female athletes have 2-8× higher risk than male athletes in comparable sports (biomechanical, hormonal, anatomic factors).
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Risk factors
- Pivoting sports — soccer, basketball, football, skiing, lacrosse
- Female sex (in pivoting sports)
- Narrow intercondylar notch
- Increased posterior tibial slope
- Quadriceps-dominant landing biomechanics
- Generalized ligamentous laxity
- Prior ACL injury (10-25% retear or contralateral tear within 5 years)
- Inadequate neuromuscular training
Pathophysiology
Non-contact mechanism: sudden deceleration, valgus collapse, and tibial internal rotation with the knee near full extension. The ACL fails as anterior tibial translation exceeds its tensile strength. Up to 60% of ACL tears have associated meniscal injury; chronic ACL deficiency predisposes to early osteoarthritis even after reconstruction.
Clinical presentation
Symptoms
- Non-contact pivoting or deceleration injury with audible/sensed 'pop'
- Immediate large effusion (within 1-2 hours) — hemarthrosis
- Inability to continue activity
- Knee instability ('giving way') with subsequent pivots
- Pain often diffuse
Signs / physical exam
- Large effusion
- Lachman test — most sensitive (knee 20-30° flexion, anterior translation of tibia, soft endpoint)
- Anterior drawer test (90° flexion)
- Pivot-shift test — most specific but uncomfortable and often requires anesthesia for reliable performance
- Range of motion often limited by effusion and pain
- Examine for associated MCL injury (valgus stress), meniscus tear (joint-line tenderness)
Differential diagnosis
- Meniscus tear — Joint-line pain, slower effusion (12-24 h), mechanical symptoms; often coexists
- MCL sprain — Valgus mechanism, medial pain, pain with valgus stress; isolated tear has smaller effusion
- PCL tear — Posterior tibial translation, dashboard mechanism, positive posterior drawer
- Patellar dislocation — Lateral displacement of patella, hemarthrosis, positive apprehension; can mimic ACL by mechanism
- Tibial plateau fracture — High-energy mechanism, X-ray findings, sometimes subtle (lipohemarthrosis on lateral view)
- Quadriceps or patellar tendon rupture — Inability to extend knee against gravity, palpable defect
Diagnostic workup
Labs
- Not indicated
Imaging
- Knee X-rays — evaluate for fractures, including Segond fracture (avulsion of lateral capsule from the lateral tibial plateau — pathognomonic for ACL tear)
- MRI — gold standard; confirms diagnosis, characterizes associated injuries (meniscus, MCL, bone bruises in classic 'kissing contusion' pattern at lateral femoral condyle and posterior lateral tibial plateau)
- Arthrocentesis — bloody fluid (hemarthrosis); presence of fat globules suggests intra-articular fracture
Diagnostic algorithm
flowchart TD
A[Knee twisting injury<br/>pop + immediate effusion] --> B[Knee X-rays<br/>look for Segond fracture]
B --> C[Lachman test<br/>anterior drawer, pivot-shift]
C --> D{Lachman<br/>positive?}
D -->|Yes| E[MRI confirms ACL tear<br/>characterize associated injuries]
D -->|No, equivocal| F[MRI for definitive dx]
E --> G[RICE, motion, PT to restore extension]
G --> H{Active patient<br/>or pivoting sport?}
H -->|Yes| I[ACL reconstruction<br/>after motion restored]
H -->|No| J[Non-operative care<br/>+ functional bracing]
I --> K[9-12 mo rehab to RTS]
J --> KComplications
- Persistent instability if non-operative care chosen and patient returns to pivoting sport
- Post-traumatic osteoarthritis (high risk regardless of surgical or non-operative management)
- Meniscal tear progression with ongoing instability
- Graft failure or retear (~5-15%, higher in young athletes returning to sport)
- Arthrofibrosis if early surgery before motion restored
- DVT/PE (low)
- Postoperative infection
- Contralateral ACL injury (similar high risk)
PANCE pearls
- Immediate large effusion + audible pop + pivoting mechanism is ACL until proven otherwise.
- Segond fracture on plain radiograph is pathognomonic for ACL injury.
- Lachman test is more sensitive than anterior drawer; pivot-shift is more specific.
- Restore full extension and reduce swelling BEFORE surgical reconstruction to minimize arthrofibrosis.
- ACL reconstruction does not prevent post-traumatic osteoarthritis — counsel patients realistically.
- Neuromuscular training programs (e.g., PEP) reduce ACL injury risk in female athletes.
References
- AAOS 2022 — AAOS Clinical Practice Guideline on Management of Anterior Cruciate Ligament Injuries (2022)
- MOON Group — Multicenter Orthopaedic Outcomes Network (MOON) Knee Cohort — long-term outcomes of ACL reconstruction (Spindler et al., AJSM)
- BEAR Trial — Bridge-Enhanced ACL Repair vs Reconstruction (Murray et al., AJSM 2020)
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