Neurology · PANCE / PANRE

Subarachnoid Hemorrhage (SAH)

Bleeding into the subarachnoid space, usually from a ruptured saccular aneurysm.

Also known as: SAH, aneurysmal subarachnoid hemorrhage, thunderclap headache

Overview

Acute bleeding into the subarachnoid space between the arachnoid and pia mater. Most cases (~85%) are due to rupture of a saccular ('berry') aneurysm; others are perimesencephalic, traumatic, or from AVM, vasculitis, or coagulopathy.

Epidemiology

Incidence ~9/100,000 person-years. Peak age 40-60. Female predominance (3:2). Mortality 25-50% overall; ~10-15% die before reaching hospital.

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Risk factors

  • Hypertension
  • Smoking
  • Heavy alcohol use
  • Sympathomimetic drug use (cocaine, methamphetamine)
  • Family history of SAH (first-degree relative)
  • Connective tissue disorders: autosomal dominant polycystic kidney disease, Ehlers-Danlos type IV, Marfan, neurofibromatosis
  • Female sex, especially post-menopausal

Pathophysiology

Most saccular aneurysms arise at branch points of the circle of Willis (anterior communicating artery most common, then posterior communicating, then MCA bifurcation). Aneurysm rupture releases arterial blood into the subarachnoid cisterns, causing sudden severe headache, transient global cerebral ischemia, and elevated ICP. Subsequent complications (rebleeding, vasospasm, hydrocephalus, seizures) determine outcome.

Clinical presentation

Symptoms

  • Sudden severe headache ('thunderclap,' 'worst headache of life,' maximal within seconds)
  • Brief loss of consciousness in ~50%
  • Nausea, vomiting, photophobia
  • Meningismus develops over hours from blood irritating meninges
  • Sentinel headache in days-weeks before rupture in 10-40% (warning leak)
  • Seizures at onset in ~10%

Signs / physical exam

  • Meningismus (Kernig and Brudzinski signs)
  • Decreased level of consciousness
  • Focal deficits: CN III palsy (posterior communicating aneurysm — ipsilateral fixed dilated pupil), hemiparesis (vasospasm or hematoma)
  • Subhyaloid (preretinal) hemorrhages on funduscopy (Terson syndrome)
  • Elevated BP and pulse pressure

Classic findings

Thunderclap headache + meningismus + LOC. Hunt-Hess and World Federation of Neurosurgical Societies (WFNS) grading scales predict outcome.

Differential diagnosis

  • Primary thunderclap headache / RCVS (reversible cerebral vasoconstriction) — Recurrent thunderclap headaches over days-weeks; segmental vasoconstriction on angiography reverses by 12 weeks
  • Migraine — Usually slower onset, history of similar episodes; SAH must be excluded for first severe headache
  • Meningitis — Fever, photophobia, nuchal rigidity, gradual onset; CSF pleocytosis with no xanthochromia
  • Cervical artery dissection — Neck or facial pain, partial Horner syndrome (carotid) or posterior circulation symptoms (vertebral); CTA/MRA diagnostic
  • Pituitary apoplexy — Severe headache + visual field defect + cranial nerve palsies + endocrine dysfunction; sellar imaging
  • Cerebral venous sinus thrombosis — Subacute progressive headache, papilledema, seizures, focal deficits; CTV/MRV with empty delta sign
  • Hypertensive emergency — BP often >220/120; headache improves with BP control; CT negative for blood

Diagnostic workup

Diagnostic criteria

Subarachnoid blood on CT in basal cisterns, sylvian fissure, or interhemispheric fissure; or xanthochromia on LP.

Labs

  • CBC, coagulation panel, BMP, troponin (neurogenic stunned myocardium common)
  • Type and screen

Imaging

  • Non-contrast head CT within 6 h of symptom onset is ~100% sensitive (drops to ~85% at 24 h, ~50% at 1 week)
  • Lumbar puncture if CT negative but clinical suspicion remains — xanthochromia (yellow CSF from hemoglobin breakdown) confirms SAH; RBCs that don't clear between tubes 1 and 4
  • CT angiography head/neck — identifies aneurysm location, size, neck for treatment planning
  • Catheter angiography (DSA) — gold standard, used during coiling
  • Repeat MRI/MRA or DSA in 1-2 weeks if initial study negative but SAH confirmed

Diagnostic algorithm

Hunt-Hess GradeClinical FindingsMortality
IAsymptomatic or minimal headache, mild nuchal rigidity~1%
IIModerate-severe headache, nuchal rigidity, no deficit except cranial nerve palsy~5%
IIIDrowsy, confused, mild focal deficit~19%
IVStupor, moderate-severe hemiparesis, early decerebrate posturing~42%
VDeep coma, decerebrate posturing, moribund~77%
Hunt-Hess grading scale for aneurysmal subarachnoid hemorrhage.

Treatment

First-line

  • ABCs; intubate if GCS ≤8 or airway compromise
  • BP control: target SBP <160 (or MAP <110) until aneurysm secured — IV nicardipine, clevidipine, or labetalol
  • Pain control (acetaminophen, opioids; avoid antiplatelet/NSAID)
  • Antiemetics, stool softeners (avoid Valsalva)
  • Nimodipine 60 mg PO/NG q4h × 21 days — improves neurologic outcomes by reducing vasospasm-related ischemia (not by preventing vasospasm itself)
  • Definitive aneurysm treatment within 24-72 h: endovascular coiling (preferred when feasible — ISAT) or surgical clipping
  • Seizure prophylaxis (short-course levetiracetam) controversial; many centers use only with documented seizures

Second-line / adjunct

  • External ventricular drain for hydrocephalus or to monitor ICP
  • Transcranial doppler daily to monitor for vasospasm (peak days 4-14)
  • Vasospasm management: induced hypertension (after aneurysm secured), intra-arterial vasodilators (verapamil, nicardipine), or angioplasty
  • DVT prophylaxis (mechanical until aneurysm secured, then chemical)

Complications

  • Rebleeding (highest risk first 24 h; ~40% mortality) — prevent by securing aneurysm early
  • Cerebral vasospasm and delayed cerebral ischemia (days 4-14) — leading cause of morbidity in survivors
  • Hydrocephalus (acute communicating or obstructive; chronic later)
  • Hyponatremia from cerebral salt wasting or SIADH
  • Neurogenic pulmonary edema, stunned myocardium, arrhythmias
  • Seizures (early or late)

PANCE pearls

  • CT within 6 hours of headache onset, interpreted by an experienced reader, is essentially 100% sensitive — LP may be safely omitted in this setting (Perry rules).
  • Xanthochromia takes 6-12 hours to develop — LP done too early may miss SAH.
  • Posterior communicating artery aneurysm → ipsilateral CN III palsy with pupil involvement (compressive); diabetic CN III spares the pupil.
  • ISAT trial: coiling was superior to clipping for survival and functional independence in ruptured anterior circulation aneurysms.
  • Nimodipine improves outcomes but does NOT reliably prevent angiographic vasospasm — mechanism likely neuroprotective.

References

  • AHA/ASA 2023 — 2023 Guideline for the Management of Patients With Aneurysmal Subarachnoid Hemorrhage (Hoh et al., Stroke 2023)
  • ISAT — International Subarachnoid Aneurysm Trial: Coiling versus Clipping (Molyneux et al., Lancet 2002, 2005)
  • Perry Rules — Clinical Decision Rules to Rule Out Subarachnoid Hemorrhage for Acute Headache (Perry et al., JAMA 2013)
  • Hunt-Hess — Surgical Risk as Related to Time of Intervention in Repair of Intracranial Aneurysms (Hunt & Hess, J Neurosurg 1968)

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