Gastrointestinal · PANCE / PANRE

Choledocholithiasis and Ascending Cholangitis

CBD stones cause obstructive jaundice; superimposed infection produces ascending cholangitis.

Also known as: choledocholithiasis, CBD stones, ascending cholangitis, Charcot triad, Reynolds pentad

Overview

Choledocholithiasis: gallstones in the common bile duct (CBD). Ascending cholangitis: bacterial infection of the biliary tree, typically resulting from biliary obstruction (stone, stricture, malignancy) and ascending bacterial colonization, presenting with Charcot triad (fever, jaundice, RUQ pain) or Reynolds pentad (Charcot + hypotension + altered mental status).

Epidemiology

Choledocholithiasis present in 10-20% of patients with gallstones. Cholangitis affects 1-9% of patients with CBD stones; mortality 5-10% with prompt treatment, up to 50% if untreated or with severe sepsis.

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Risk factors

  • Cholelithiasis (primary risk factor)
  • Prior cholecystectomy with retained or de novo CBD stones
  • Asian biliary parasitic infection (Clonorchis, Ascaris) → brown pigment stones
  • Biliary strictures (PSC, post-surgical, malignant)
  • Indwelling biliary stents
  • Choledochal cysts
  • Biliary anomalies

Pathophysiology

CBD stone obstructs flow → biliary stasis and increased pressure → bacterial proliferation (E. coli, Klebsiella, Enterobacter, Enterococcus, anaerobes) → ascending infection → bacteremia and sepsis. Increased intrabiliary pressure drives translocation of bacteria into the bloodstream.

Clinical presentation

Symptoms

  • Choledocholithiasis: RUQ pain, jaundice, dark urine, pale stools, pruritus
  • Cholangitis (Charcot triad): fever, RUQ pain, jaundice
  • Cholangitis (Reynolds pentad): Charcot triad + hypotension + altered mental status (severe/suppurative)
  • Nausea, vomiting
  • Rigors

Signs / physical exam

  • Jaundice, scleral icterus
  • RUQ tenderness
  • Fever, tachycardia
  • Hypotension if severe (Reynolds pentad)
  • Altered mental status — sepsis-induced encephalopathy
  • Excoriations from pruritus
  • Hepatomegaly possible

Classic findings

Charcot triad (fever + RUQ pain + jaundice) in any patient with gallstones — assume cholangitis until proven otherwise; this is a surgical/endoscopic emergency.

Differential diagnosis

  • Acute cholecystitis without CBD involvement — Murphy sign, gallbladder wall thickening on US, normal CBD diameter, mildly elevated LFTs only
  • Hepatitis (viral, alcoholic, drug-induced) — Markedly elevated AST/ALT, viral serologies; alk phos and bili less elevated proportionally
  • Pancreatic head malignancy — Painless jaundice, Courvoisier sign (palpable nontender gallbladder), weight loss; mass on imaging
  • Cholangiocarcinoma — Painless jaundice, irregular biliary stricture on MRCP/EUS
  • Primary sclerosing cholangitis — Beaded biliary strictures on MRCP; IBD association; pANCA+
  • Biliary stricture (post-surgical, ischemic) — Prior cholecystectomy, transplant
  • Mirizzi syndrome — Cystic duct stone compressing CBD; MRCP/EUS
  • Acute pancreatitis — Pain to back, lipase >3× ULN — may coexist with biliary obstruction

Diagnostic workup

Diagnostic criteria

ASGE risk stratification for CBD stones (2019): High probability (any 1 of: CBD stone on imaging; total bilirubin >4 mg/dL WITH a dilated CBD; or clinical ascending cholangitis) → proceed to ERCP. Intermediate (CBD dilation + elevated LFTs + bilirubin 1.8-4) → MRCP or EUS. Low → cholecystectomy alone. Tokyo Guidelines TG18 for cholangitis: A) systemic inflammation (fever, leukocytosis, CRP); B) cholestasis (jaundice, abnormal LFTs); C) imaging (biliary dilation, evidence of cause). Diagnosis requires 1 from each category.

Labs

  • CBC — leukocytosis with left shift
  • BMP — assess for AKI in sepsis
  • LFTs — elevated direct bilirubin, alk phos, GGT (cholestatic pattern); AST/ALT also elevated
  • Lipase — exclude or identify concurrent pancreatitis
  • Coagulation studies — INR may rise (vitamin K malabsorption in prolonged cholestasis)
  • Blood cultures × 2 — guide antibiotic therapy
  • Lactate, procalcitonin if septic

Imaging

  • RUQ ultrasound — first-line; identifies gallstones, dilated CBD (>6 mm in patients <60, >8 mm post-cholecystectomy), but sensitivity for CBD stones only 20-50%
  • MRCP — non-invasive, high sensitivity (>90%) for CBD stones
  • EUS — most sensitive for small CBD stones; preferred when MRCP equivocal
  • ERCP — diagnostic AND therapeutic; reserved for high-probability or therapeutic intent (do not use diagnostically due to pancreatitis risk)
  • CT — alternative when malignancy suspected; less sensitive for non-calcified stones

Diagnostic algorithm

flowchart TD
  A[Suspected biliary disease<br/>RUQ pain + jaundice] --> B[Labs + RUQ US]
  B --> C{Charcot triad?<br/>Fever + RUQ pain<br/>+ jaundice}
  C -->|Yes — cholangitis| D[IV fluids + broad-spectrum<br/>antibiotics<br/>URGENT ERCP within 24h]
  C -->|No| E{CBD stone probability<br/>ASGE 2019}
  E -->|High: CBD stone seen<br/>or bili >4 + clinical cholangitis| F[ERCP with sphincterotomy<br/>+ stone extraction]
  E -->|Intermediate| G[MRCP or EUS]
  G --> H{Stone present?}
  H -->|Yes| F
  H -->|No| I[Laparoscopic cholecystectomy<br/>± intraop cholangiogram]
  E -->|Low| I
  D --> J[Cholecystectomy after recovery]
  F --> J
Choledocholithiasis and cholangitis pathway — ASGE 2019 risk stratification and Tokyo Guidelines management.

Treatment

First-line

  • Resuscitation: IV fluids, hemodynamic support
  • Broad-spectrum IV antibiotics covering Gram-negative and anaerobes — piperacillin-tazobactam, ceftriaxone + metronidazole, or carbapenem for severe sepsis/multi-drug-resistant risk
  • URGENT biliary drainage by ERCP with sphincterotomy and stone extraction — within 24-48 h for moderate cholangitis, within 24 h for severe cholangitis
  • Percutaneous transhepatic cholangiography (PTC) drainage if ERCP fails or contraindicated (post-Roux-en-Y anatomy)
  • Surgical decompression (open CBD exploration) — rarely required as rescue
  • Cholecystectomy after recovery (same admission or within 4-6 weeks) to prevent recurrence

Choledocholithiasis WITHOUT cholangitis (high probability of stones)

  • ERCP with sphincterotomy and stone extraction PRIOR to cholecystectomy (preferred)
  • Or laparoscopic cholecystectomy with intraoperative cholangiography and laparoscopic CBD exploration

Ascending cholangitis — TG18 severity

  • Grade I (mild): IV antibiotics + non-urgent biliary drainage within 24-48 h
  • Grade II (moderate): early biliary drainage (within 24 h) + antibiotics
  • Grade III (severe — organ dysfunction): emergent drainage + ICU-level care + antibiotics; mortality remains 10-20%

Second-line / adjunct

  • EUS-guided biliary drainage if ERCP fails
  • Mechanical lithotripsy, balloon dilation, or large-balloon sphincteroplasty for large or difficult stones
  • Biliary stent placement if stones cannot be extracted at first ERCP (allows decompression and drainage; second-look ERCP later)
  • Source control of malignant obstruction with stent (plastic or self-expanding metal)

Complications

  • Septic shock, multiorgan failure
  • Hepatic abscess
  • Acute pancreatitis (post-ERCP or stone-induced)
  • Bile peritonitis (post-procedure)
  • Recurrent stones
  • Secondary biliary cirrhosis (chronic obstruction)
  • Biliary stricture
  • Cholangiocarcinoma (chronic biliary inflammation, especially with parasitic infection — recurrent pyogenic cholangitis)

PANCE pearls

  • Charcot triad (fever + RUQ pain + jaundice) = ascending cholangitis until proven otherwise; Reynolds pentad adds hypotension and altered mental status (severe).
  • Urgent biliary drainage is the cornerstone of cholangitis treatment — antibiotics alone are insufficient.
  • ERCP is therapeutic (sphincterotomy + stone extraction) and should be used selectively given pancreatitis risk; for diagnosis, use MRCP or EUS.
  • Rectal indomethacin reduces post-ERCP pancreatitis risk in high-risk patients (Elmunzer, NEJM 2012).
  • Same-admission cholecystectomy after CBD stone extraction reduces readmission and complications (PONCHO trial).
  • Painless jaundice with palpable distended gallbladder (Courvoisier sign) suggests malignancy (pancreatic head cancer, cholangiocarcinoma) — NOT stones.
  • Brown pigment stones in Asian patients suggest biliary parasitic infection (Clonorchis, Ascaris) and chronic recurrent cholangitis.
  • Post-cholecystectomy CBD diameter is normally larger (up to 10 mm) — recalibrate sonographic thresholds.
  • Mirizzi syndrome: external compression of CBD by cystic duct stone — preoperative MRCP critical to avoid bile duct injury.

References

  • Tokyo Guidelines TG18 — Kiriyama S et al. Tokyo Guidelines 2018: diagnostic criteria and severity grading of acute cholangitis. J Hepatobiliary Pancreat Sci 2018;25:17-30
  • ASGE 2019 — ASGE Standards of Practice Committee. ASGE guideline on the role of endoscopy in the evaluation and management of choledocholithiasis. Gastrointest Endosc 2019;89:1075-1105
  • PONCHO Trial — da Costa DW et al. Same-admission versus interval cholecystectomy for mild gallstone pancreatitis (PONCHO). Lancet 2015;386:1261-1268
  • Rectal Indomethacin — Elmunzer BJ et al. A Randomized Trial of Rectal Indomethacin to Prevent Post-ERCP Pancreatitis. NEJM 2012;366:1414-1422

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