Gastrointestinal · PANCE / PANRE

Irritable Bowel Syndrome (IBS)

Disorder of gut-brain interaction with recurrent abdominal pain and altered bowel habits without structural disease.

Also known as: IBS, irritable bowel syndrome, IBS-D, IBS-C, IBS-M, functional bowel disorder

Overview

A disorder of gut-brain interaction characterized by recurrent abdominal pain associated with defecation or change in bowel habits, in the absence of identifiable structural, biochemical, or inflammatory abnormalities. Diagnosed by Rome IV criteria.

Epidemiology

Prevalence 5-15% in Western countries; 2:1 female-to-male predominance; typical onset under age 50. Major contributor to outpatient GI visits and loss of work productivity.

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Risk factors

  • Female sex, younger age
  • Prior gastrointestinal infection (post-infectious IBS — Campylobacter, Salmonella, Giardia, COVID-19)
  • Psychiatric comorbidity: anxiety, depression, somatization, prior trauma/abuse
  • Family history
  • Small intestinal bacterial overgrowth (SIBO) overlap
  • Food sensitivities (FODMAPs)

Pathophysiology

Multifactorial: altered gut motility, visceral hypersensitivity, dysregulated brain-gut axis, altered microbiome, mucosal immune activation, increased intestinal permeability, and bile acid malabsorption. Stress and emotion modulate symptom severity.

Clinical presentation

Symptoms

  • Recurrent abdominal pain related to defecation (relief or worsening)
  • Change in stool frequency or form
  • Bloating, distension
  • Mucus in stool
  • Symptoms worsened by stress and certain foods (FODMAPs)
  • Sensation of incomplete evacuation

Signs / physical exam

  • Normal physical exam
  • Mild abdominal tenderness sometimes
  • ABSENCE of alarm features: no weight loss, no GI bleeding, no anemia, no nocturnal symptoms, no fever, no palpable mass

Classic findings

Young woman with chronic abdominal pain relieved by defecation, alternating bowel habit, and bloating — exam normal.

Differential diagnosis

  • Inflammatory bowel disease (Crohn, UC) — Bloody diarrhea, nocturnal symptoms, weight loss, fever, elevated CRP/calprotectin; colonoscopy with biopsy
  • Celiac disease — Diarrhea, bloating, anemia, dermatitis herpetiformis; TTG IgA and total IgA; biopsy
  • Microscopic colitis — Chronic watery diarrhea in older women; colonoscopy NORMAL — diagnosis by random biopsies
  • Lactose intolerance / fructose malabsorption — Symptom-trigger relation; trial of elimination diet or breath test
  • SIBO — Bloating, diarrhea or constipation; glucose or lactulose breath test; treat with rifaximin
  • Bile acid diarrhea — Watery diarrhea after cholecystectomy or terminal ileal disease; SeHCAT or empiric cholestyramine trial
  • Colorectal cancer — Older age, weight loss, anemia, GI bleeding, family history; colonoscopy
  • Hyperthyroidism (diarrhea) / hypothyroidism (constipation) — Systemic features; TSH
  • Chronic pancreatitis — Steatorrhea, weight loss, calcifications; fecal elastase
  • Ovarian cancer — Bloating in women >50; pelvic exam, CA-125, ultrasound

Diagnostic workup

Diagnostic criteria

Rome IV: Recurrent abdominal pain on average ≥1 day/week in the last 3 months, associated with ≥2 of: (1) related to defecation, (2) change in stool frequency, (3) change in stool form. Symptom onset ≥6 months prior to diagnosis. Subtypes by predominant stool form using Bristol Stool Scale: IBS-C, IBS-D, IBS-M, IBS-U.

Labs

  • CBC (exclude anemia)
  • CRP (exclude inflammation)
  • Fecal calprotectin (helpful to exclude IBD if borderline)
  • TTG IgA + total IgA (exclude celiac)
  • TSH (exclude thyroid disease)
  • Stool studies (Giardia antigen, C. diff, ova/parasites) if recent infection or travel

Imaging

  • Colonoscopy — NOT routinely required for typical IBS without alarm features; perform if age ≥45 (CRC screening), alarm features, family history of IBD/CRC, or refractory symptoms
  • Random colonic biopsies if chronic watery diarrhea (microscopic colitis)

Diagnostic algorithm

IBS SubtypeBristol Stool Scale PatternPreferred Pharmacotherapy
IBS-C>25% type 1-2, <25% type 6-7Linaclotide, plecanatide, lubiprostone, PEG
IBS-D>25% type 6-7, <25% type 1-2Rifaximin, loperamide, eluxadoline, alosetron
IBS-M>25% both ends of spectrumTailor by predominant symptom; TCAs for pain
IBS-UInsufficient abnormality to classifySymptom-targeted
Rome IV IBS subtypes by Bristol Stool Scale and first-line pharmacotherapy.

Treatment

First-line

  • Establish therapeutic relationship; educate that IBS is a real, chronic, manageable condition
  • Dietary modification: regular meals, adequate fiber (soluble — psyllium); trial of low-FODMAP diet under dietitian guidance
  • Exercise, sleep hygiene, stress management
  • Cognitive behavioral therapy and gut-directed hypnotherapy (strong evidence)

IBS-C (constipation predominant)

  • Soluble fiber (psyllium)
  • PEG 3350
  • Linaclotide, plecanatide, or lubiprostone (prosecretory)
  • Tenapanor (NHE3 inhibitor)
  • Avoid stimulant laxatives long-term

IBS-D (diarrhea predominant)

  • Loperamide PRN
  • Rifaximin 550 mg TID × 14 days (can repeat up to twice for recurrence)
  • Bile acid sequestrants — cholestyramine, colesevelam — if bile acid diarrhea suspected
  • Eluxadoline (mu/kappa-opioid agonist, delta antagonist) — avoid post-cholecystectomy and in heavy drinkers (sphincter of Oddi spasm)
  • Alosetron — 5-HT3 antagonist; restricted use in women with severe refractory IBS-D

IBS-M (mixed)

  • Tailor to predominant symptom at the time
  • TCAs particularly useful for pain-predominant phenotypes

Second-line / adjunct

  • Symptom-based pharmacotherapy (see by_subtype)
  • Tricyclic antidepressants (amitriptyline, nortriptyline, desipramine) — pain and IBS-D; low dose 10-50 mg at bedtime
  • SSRIs (citalopram, sertraline, paroxetine) — global symptoms with comorbid mood disorder
  • Antispasmodics — dicyclomine, hyoscyamine, peppermint oil — for cramping abdominal pain

Complications

  • Impaired quality of life and work productivity
  • Anxiety, depression, somatization (often pre-existing)
  • Healthcare overutilization, repeated testing
  • Side effects of long-term laxative or opioid use
  • Diagnostic uncertainty leading to missed structural disease (if alarm features ignored)

PANCE pearls

  • Diagnose IBS POSITIVELY using Rome IV criteria — avoid diagnosis of exclusion approach with exhaustive testing.
  • Alarm features mandating colonoscopy: age ≥45 (CRC screening), GI bleeding, unexplained anemia, weight loss, nocturnal symptoms, family history of IBD or CRC, palpable mass.
  • Fecal calprotectin <50 mcg/g effectively excludes active IBD; >250 strongly suggests IBD.
  • Low-FODMAP diet has strong evidence but should be supervised by a dietitian — strict phase ≤6 weeks, followed by reintroduction.
  • Rifaximin is non-absorbable and effective for IBS-D bloating; up to 3 courses approved for recurrence.
  • Linaclotide and plecanatide are guanylate cyclase-C agonists — improve both constipation and IBS-related pain.
  • Avoid chronic opioid use — opioid-induced constipation and narcotic bowel syndrome amplify IBS symptoms.
  • CBT and gut-directed hypnotherapy have effect sizes rivaling pharmacotherapy for refractory IBS.

References

  • ACG 2021 — Lacy BE et al. ACG Clinical Guideline: Management of Irritable Bowel Syndrome. Am J Gastroenterol 2021;116:17-44
  • AGA 2022 — Chang L et al. AGA Clinical Practice Guideline on the Pharmacological Management of IBS with Constipation and Diarrhea. Gastroenterology 2022;163:118-160
  • Rome IV — Lacy BE et al. Bowel Disorders. Gastroenterology 2016;150:1393-1407

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