Pulmonary · PANCE / PANRE

Acute Bronchitis

Self-limited inflammation of the large airways, almost always viral.

Also known as: acute bronchitis, viral bronchitis, chest cold

Overview

Self-limited inflammation of the trachea and large bronchi, predominantly viral in etiology, characterized by acute cough lasting up to 3 weeks, with or without sputum production, in patients without chronic lung disease.

Epidemiology

One of the most common reasons for ambulatory visits in the US (~5% of adults annually). Peak incidence in fall and winter. Antibiotic overprescription is a major stewardship concern — ~70% of cases historically received antibiotics despite minimal benefit.

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Risk factors

  • Recent viral upper respiratory infection
  • Smoking, secondhand smoke
  • Environmental irritants (cold air, allergens, fumes)
  • Air pollution
  • Crowded living conditions, daycare exposure

Pathophysiology

Viral infection of bronchial epithelium → mucosal inflammation, edema, hypersecretion of mucus, bronchial hyperresponsiveness, and sloughing of cells → cough that can persist for 1-3 weeks after acute infection resolves due to lingering airway inflammation. Common pathogens: influenza A and B, RSV, rhinovirus, coronavirus (including SARS-CoV-2), adenovirus, parainfluenza, metapneumovirus. Bacterial causes (<10%): Bordetella pertussis, Mycoplasma pneumoniae, Chlamydophila pneumoniae.

Clinical presentation

Symptoms

  • Cough — initially dry, often becoming productive of clear or purulent sputum (PURULENT SPUTUM ALONE DOES NOT INDICATE BACTERIAL INFECTION)
  • Mild dyspnea, wheeze, chest discomfort with coughing
  • Often preceded by URI symptoms: rhinorrhea, sore throat, mild fever (low-grade)
  • Cough typically resolves within 1-3 weeks; postinfectious cough can last 6-8 weeks

Signs / physical exam

  • Generally normal exam; possible rhonchi/wheeze that clear with coughing
  • Low-grade fever or afebrile
  • Normal SpO2 and respiratory rate
  • Absence of focal consolidation, dullness, egophony (would suggest pneumonia)

Differential diagnosis

  • Community-acquired pneumonia — Fever >38°C, tachycardia, tachypnea, focal exam findings, abnormal CXR with infiltrate
  • Influenza — Abrupt fevers, myalgia, headache, seasonal occurrence; positive rapid antigen/PCR; tamiflu within 48 h
  • COVID-19 — Anosmia, ageusia, contacts, positive PCR/antigen; can have prolonged cough
  • Pertussis — Paroxysmal coughing fits with inspiratory whoop, post-tussive emesis, >2 weeks of cough; nasopharyngeal PCR
  • Asthma exacerbation — Wheeze, prior asthma history, reversibility with bronchodilator
  • GERD-related cough — Cough worse supine, postprandial, regurgitation; responds to PPI trial
  • Postnasal drip / upper airway cough syndrome — Throat clearing, drip sensation, allergic rhinitis; responds to antihistamines/nasal steroids
  • ACE inhibitor cough — Dry cough developing after ACEi start; resolves on discontinuation (may take 4 weeks)

Diagnostic workup

Labs

  • Generally NONE needed in immunocompetent ambulatory patients with classic presentation
  • Procalcitonin and CRP not routinely indicated but can support antibiotic stewardship
  • Targeted viral testing: influenza (if antivirals considered or seasonal/outbreak), SARS-CoV-2 (per current public health guidance), RSV in select populations
  • Pertussis PCR (nasopharyngeal) if classic paroxysmal cough with whoop or exposure

Imaging

  • Chest radiograph NOT routinely needed — obtain if any of: HR >100, RR >24, T >38°C, focal exam findings (consolidation, rales), age >65, or immunocompromise — to exclude pneumonia
  • CDC/IDSA suggest CXR to differentiate from CAP when clinical picture is uncertain

Diagnostic algorithm

FeatureAcute BronchitisPneumonia
VitalsOften normalFever >38°C, HR >100, RR >24
ExamDiffuse rhonchi/wheeze; no consolidationFocal crackles, egophony, dullness
CXRNormal — usually not neededLobar or interstitial infiltrate
TreatmentSymptomatic onlyAntibiotic therapy
CourseResolves in 1-3 weeksImproves with appropriate antibiotics
Bedside differentiation of acute bronchitis from community-acquired pneumonia.

Treatment

First-line

  • Symptomatic and supportive care — antibiotics generally NOT indicated (high-value care)
  • Hydration, rest
  • Antitussives (dextromethorphan, benzonatate) for bothersome cough (modest evidence)
  • Honey 1-2 tsp (for adults and children ≥1 year) — evidence-based cough relief, particularly nocturnal
  • Throat lozenges, humidified air
  • Inhaled short-acting beta-agonist (albuterol) reasonable if wheeze or bronchospasm; not for all comers
  • NSAIDs or acetaminophen for fever or myalgia
  • Influenza-positive: oseltamivir 75 mg BID × 5 days if within 48 h of onset or high-risk patient

Second-line / adjunct

  • Antibiotics ONLY for documented or strongly suspected bacterial cause:
  • Pertussis (high public health priority): macrolide — azithromycin 500 mg day 1 then 250 mg × 4 days OR clarithromycin; primary benefit is reduced transmission
  • Mycoplasma or Chlamydophila (uncommon in true acute bronchitis): macrolide or doxycycline
  • Patient education on expected duration (cough up to 3 weeks; postinfectious up to 6-8 weeks) to reduce antibiotic-seeking
  • Re-evaluate if symptoms persist >3 weeks or worsen — consider pertussis, postnasal drip, asthma, GERD, ACEi cough, or alternative diagnosis

Complications

  • Postinfectious cough (up to 6-8 weeks)
  • Bronchial hyperreactivity / transient asthma-like symptoms
  • Superimposed bacterial pneumonia (uncommon)
  • Exacerbation of underlying COPD/asthma if unrecognized chronic disease
  • Pertussis sequelae in vulnerable contacts (especially infants)

PANCE pearls

  • Purulent (yellow/green) sputum does NOT indicate bacterial infection in acute bronchitis — color reflects neutrophilic inflammation, not pathogen.
  • Routine antibiotic use does not shorten illness; ACP/CDC have campaigned for stewardship in this setting.
  • Obtain CXR if HR >100, RR >24, T >38°C, age >65, or focal exam to exclude pneumonia.
  • Persistent cough >3 weeks should prompt evaluation for pertussis, asthma, postnasal drip, GERD, or ACEi cough.
  • Honey is the only widely accepted symptomatic intervention with evidence in both adults and children ≥1 year (botulism risk under 1).

References

  • ACP/CDC 2016 — Appropriate Antibiotic Use for Acute Respiratory Tract Infection in Adults (Harris et al., Ann Intern Med 2016)
  • CHEST 2006 — Diagnosis and Management of Cough Executive Summary: ACCP Evidence-Based Clinical Practice Guidelines (Irwin et al., Chest 2006)
  • Cochrane Bronchitis — Antibiotics for Acute Bronchitis (Smith et al., Cochrane Database Syst Rev 2017)
  • CDC Pertussis — Recommended Antimicrobial Agents for the Treatment and Postexposure Prophylaxis of Pertussis (Tiwari et al., MMWR 2005)

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