Infectious Disease · PANCE / PANRE

Strongyloidiasis

Soil-transmitted nematode (Strongyloides stercoralis) causing chronic GI/skin disease and life-threatening hyperinfection in immunocompromised hosts.

Also known as: Strongyloides stercoralis, strongyloidiasis, hyperinfection syndrome, threadworm

Overview

Infection by the soil-transmitted intestinal nematode Strongyloides stercoralis. Unique among helminths in its capacity for autoinfection, allowing infection to persist for decades and to cause overwhelming hyperinfection syndrome in immunocompromised hosts.

Epidemiology

Endemic to tropical and subtropical regions worldwide, with foci in the southeastern United States (Appalachia), Caribbean, sub-Saharan Africa, Latin America, and Southeast Asia. Estimates of global prevalence range from 30 to 600 million. US veterans of World War II Pacific theater, Vietnam, and Korean conflicts remain at risk decades later due to autoinfection.

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Risk factors

  • Residence in or travel to endemic tropical/subtropical regions
  • Walking barefoot on contaminated soil
  • Immunosuppression — particularly CORTICOSTEROIDS at any dose (most important reversible risk factor for hyperinfection)
  • HTLV-1 infection (major risk factor for hyperinfection)
  • Solid organ transplantation, hematologic malignancy
  • Older age (institutionalized adults)
  • Alcoholism, malnutrition

Pathophysiology

Filariform larvae penetrate skin → migrate via venous circulation to lungs → ascend trachea → swallowed → mature in small intestine. Female worms produce rhabditiform larvae that pass in stool OR mature within the host and reinvade the intestinal mucosa or perianal skin (autoinfection). Corticosteroids accelerate autoinfection by suppressing host eosinophil response and possibly directly stimulating the parasite, leading to hyperinfection — massive larval dissemination to lungs, CNS, and other organs, often with co-translocated gram-negative bacterial sepsis.

Clinical presentation

Symptoms

  • Chronic uncomplicated infection: often asymptomatic; intermittent abdominal pain, bloating, diarrhea, nausea
  • Cutaneous larva currens — pruritic, urticarial, serpiginous, rapidly migrating rash on buttocks, perineum, and lower trunk (pathognomonic, moves cm/hour)
  • Pulmonary: cough, wheezing, transient infiltrates (Loeffler syndrome) during initial migration
  • Hyperinfection: fever, severe abdominal pain, GI bleeding, ileus, ARDS, gram-negative bacteremia and meningitis
  • Disseminated infection in immunocompromised host with extraintestinal larvae in lungs, CNS, skin, liver

Signs / physical exam

  • Eosinophilia (often the only chronic finding; ABSENT in many hyperinfection cases — ominous)
  • Urticaria, larva currens pattern
  • Wheezing, crackles in pulmonary disease
  • Peritoneal signs in severe enteritis
  • Petechiae or 'thumbprint' periumbilical purpura in disseminated disease

Classic findings

Chronic peripheral eosinophilia in an immigrant or veteran from an endemic region. Larva currens (serpiginous, rapidly migrating urticarial rash). Hyperinfection in a patient started on corticosteroids without prior screening — sometimes with concurrent gram-negative bacteremia and meningitis.

Differential diagnosis

  • Other intestinal nematode infection (ascariasis, hookworm) — Eosinophilia, anemia in hookworm, GI/respiratory symptoms in ascariasis; ova on stool ovum-and-parasite exam
  • Loeffler syndrome (transient pulmonary infiltrates with eosinophilia) — Ascaris, hookworm, Strongyloides all cause migratory larval pneumonitis
  • Hyper-eosinophilic syndrome — Sustained eosinophilia without identifiable infection; rule out parasitic disease before steroids
  • Inflammatory bowel disease — Chronic diarrhea, weight loss; endoscopy and biopsy
  • Drug reaction with eosinophilia — Temporal medication exposure; recent antibiotic or anticonvulsant
  • Toxocara, Schistosoma, filariasis — Different geography and clinical syndrome; eosinophilia in many parasitic infections
  • Gram-negative sepsis / ARDS (in hyperinfection setting) — Often coexists with hyperinfection due to translocation of gut bacteria by migrating larvae

Diagnostic workup

Diagnostic criteria

Identification of larvae in stool, sputum, BAL, or tissue is definitive. In screening before immunosuppression, positive serology is sufficient indication for empirical treatment given the consequences of missed infection.

Labs

  • Stool ovum and parasite exam — LOW sensitivity even with multiple samples (rhabditiform larvae intermittently shed)
  • Stool agar plate culture and Baermann concentration — more sensitive
  • Strongyloides serology by ELISA — most sensitive screening test; preferred for screening before immunosuppression
  • Strongyloides PCR (where available)
  • CBC with differential (eosinophilia in chronic infection; may be absent in hyperinfection)
  • BMP, LFTs
  • Sputum or BAL for larvae in hyperinfection
  • Blood cultures (gram-negative bacteremia often accompanies hyperinfection)
  • HTLV-1 serology in high-risk patients

Imaging

  • Chest x-ray and CT: bilateral infiltrates or ARDS pattern in hyperinfection
  • Abdominal imaging if ileus or perforation suspected
  • MRI brain if neurologic findings (rare disseminated CNS larvae)

Diagnostic algorithm

flowchart TD
  A[Filariform larvae<br/>penetrate skin] --> B[Venous migration to lungs<br/>Loeffler-like pneumonitis]
  B --> C[Trachea -> swallowed -><br/>small bowel adult worms]
  C --> D[Rhabditiform larvae in stool]
  D --> E[Free-living cycle in soil]
  C --> F[Autoinfection:<br/>larvae mature in host,<br/>reinvade bowel/perianal skin]
  F --> G{Immunocompromise?<br/>Steroids? HTLV-1?}
  G -->|No| H[Chronic infection<br/>eosinophilia, larva currens]
  G -->|Yes| I[Hyperinfection /<br/>dissemination<br/>ARDS, GNR sepsis]
  H --> J[Ivermectin 200 mcg/kg<br/>1-2 doses]
  I --> K[Daily ivermectin until clear<br/>+ broad-spectrum antibiotics<br/>+ reduce immunosuppression]
Strongyloides lifecycle with autoinfection and the path from chronic infection to hyperinfection.

Treatment

First-line

  • Ivermectin 200 microg/kg PO daily for 1-2 days for uncomplicated chronic infection (some clinicians repeat dose at 2 weeks)
  • Hyperinfection or disseminated disease: ivermectin 200 microg/kg/day until stool and sputum are negative for at least 2 weeks (typically 1-2+ weeks of daily dosing); subcutaneous ivermectin (veterinary product, off-label) for patients with malabsorption or ileus, in consultation with infectious diseases
  • Add empirical broad-spectrum antibacterial coverage for gram-negative enteric organisms in hyperinfection (gut translocation)
  • Reduce or discontinue corticosteroids and other immunosuppressants when possible
  • Pre-immunosuppression screening with serology in patients from endemic regions and treatment of positive results before steroid therapy or transplant

Second-line / adjunct

  • Albendazole 400 mg PO BID for 7 days — less effective than ivermectin; alternative only when ivermectin unavailable or contraindicated
  • Combination ivermectin + albendazole has been used in refractory disease
  • Repeat testing 3-6 months post-treatment to document cure (serology may decline slowly)

Complications

  • Hyperinfection syndrome with massive autoinfection and end-organ damage
  • Disseminated strongyloidiasis to lungs, CNS, liver, skin
  • Gram-negative bacteremia and meningitis from larval bowel translocation
  • ARDS, shock, multiorgan failure
  • Death rates >50% in disseminated disease despite treatment
  • Chronic malabsorption and protein-losing enteropathy

PANCE pearls

  • Screen with Strongyloides serology in any patient from an endemic area BEFORE starting corticosteroids, biologics, transplant, or chemotherapy.
  • Larva currens — rapidly migrating serpiginous rash — is highly specific for Strongyloides.
  • Eosinophilia is the chronic-infection marker but is OFTEN ABSENT in hyperinfection; do not let normal eosinophils reassure you.
  • Co-existing HTLV-1 infection markedly increases the risk of hyperinfection and reduces ivermectin efficacy.
  • Hyperinfection patients often present with concurrent gram-negative bacteremia or meningitis — empirically cover gut flora.

References

  • CDC — CDC Parasites — Strongyloides: epidemiology, diagnosis, and treatment
  • WHO — WHO Guideline on control and elimination of human schistosomiasis and soil-transmitted helminthiases (includes Strongyloides considerations)
  • IDSA / ASTMH — Practice guidelines for the management of soil-transmitted helminth infections

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